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Obesity, Glucose & InsulinWeight Gain and Metabolic DysglycemiaObesity is often triggered and perpetuated by metabolic and hormonal imbalances that create a vicious cycle of chronic dysglycemia, reduced thermogenesis, and altered fat production. This type of chronic metabolic dysregulation can make it very difficult to lose weight effectively without first addressing key functional imbalances. Glucose and insulin are primary players in the etiology of obesity. The balance between glucose and insulin in the blood can influence levels of leptin, the "hunger hormone" believed to play a strong role in regulating human appetite and metabolism. Insulin is released in response to rising blood glucose, and transports glucose to cells, where it can be used for fat production. So it comes as no surprise that upper body fat and abdominal obesity are strongly associated with "insulin resistance," a condition that arises when cell receptors resist insulin's attempt to deposit glucose, resulting in hyperinsulinemia, a state of chronically high levels of insulin. In fact, some researchers have suggested that consistently high insulin levels, both fasting and after eating, may actually instigate the metabolic process associated with obesity by increasing lipid levels in the blood. For next page, click Obesity & Glucose Obesity and Weight Loss Short Articles About Obesity and Overweight
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